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As expected, living in a city with a PFAS-contaminated water supply was associated with higher serum concentrations of PFAS chemicals. We found a significant association with three of the six PFAS chemicals evaluated (PFOA, PFNA, and PFHxS). Despite the potential for UCMR3 data to misclassify some participants—for example, those served by a small water supply not monitored under UCMR3 or by a private well, or who live in a city served by multiple public water supplies—the positive associations that we observed are consistent with findings from Hurley et al. [ 14] and emphasize that contaminated drinking water can be a substantial contributor to PFAS exposure. Having stain-resistant treated furniture and carpets were associated with higher levels of PFNA in all participants and PFDeA in non-Hispanic whites. Treated furniture and carpets are known sources of PFASs including PFNA and PFDeA [ 31], although previous studies of exposure have detected links with sulfonic, rather than carboxylic, PFASs [ 13, 52]. Haug LS, Thomsen C, Brantsaeter AL, Kvalem HE, Haugen M, Becher G, et al. Diet and particularly seafood are major sources of perfluorinated compounds in humans. Environ Int. 2010;36:772–8. Calafat AM, Wong LY, Kuklenyik Z, Reidy JA, Needham LL. Polyfluoroalkyl chemicals in the U.S. population: data from the National Health and Nutrition Examination Survey (NHANES) 2003–4 and comparisons with NHANES 1999-2000. Environ Health Perspect. 2007;115: 1596–602. Bitanihirwe BK, Lim MP, Kelley JF, Kaneko T, Woo TU. Glutamatergic deficits and parvalbumin-containing inhibitory neurons in the prefrontal cortex in schizophrenia. BMC Psychiatry. 2009;9:71. Nava N, Treccani G, Müller HK, Popoli M, Wegener G, Elfving B. The expression of plasticity-related genes in an acute model of stress is modulated by chronic desipramine in a time-dependent manner within medial prefrontal cortex. Eur Neuropsychopharmacol. 2017;27:19–28.Thirty individuals with mid-stage HD (13 men, 17 women; mean age=57.0 years, SD=10.1) were enrolled and randomly assigned to study groups. Yuen EY, Liu W, Karatsoreos IN, Ren Y, Feng J, McEwen BS, et al. Mechanisms for acute stress-induced enhancement of glutamatergic transmission and working memory. Mol Psychiatry. 2011;16:156–70. Yuen EY, Jiang Q, Chen P, Feng J, Yan Z. Activation of 5-HT2A/C receptors counteracts 5-HT1A regulation of n-methyl-D-aspartate receptor channels in pyramidal neurons of prefrontal cortex. J Biol Chem. 2008;283:17194–204. Radley JJ, Rocher AB, Miller M, Janssen WG, Liston C, Hof PR, et al. Repeated stress induces dendritic spine loss in the rat medial prefrontal cortex. Cereb Cortex. 2006;16:313–20. m and with no contraindication to exercise were recruited from the Thames valley, UK, and randomised (1:1) to intervention (exercise) or control (handwriting) groups, via a concealed computer-generated list. Groups received a 6-month, twice weekly programme. Exercise was undertaken in community facilities (30â€

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